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Elevated Basement Membrane Fibroblast Growth Factor-2 Protects Endothelial Cells in High Glucose

[+] Author Affiliations
Alisa Morss

Massachusetts Institute of Technology, Cambridge, MADrexel University, Philadelphia, PA

Michael Jonas

Massachusetts Institute of Technology, Cambridge, MATel Aviv University Sackler School of Medicine, Tel-Hashomer, Israel

Elazer R. Edelman

Massachusetts Institute of Technology, Cambridge, MA

Paper No. SBC2007-176187, pp. 995-996; 2 pages
doi:10.1115/SBC2007-176187
From:
  • ASME 2007 Summer Bioengineering Conference
  • ASME 2007 Summer Bioengineering Conference
  • Keystone, Colorado, USA, June 20–24, 2007
  • Conference Sponsors: Bioengineering Division
  • ISBN: 0-7918-4798-5
  • Copyright © 2007 by ASME

abstract

Vascular disease is the primary cause of morbidity and mortality in diabetics. Diabetic vascular disease is disseminated and includes renal capillary hypertrophy, reduced wound repair, impaired angiogenesis, and rapid and excessive hyperplasia after endovascular intervention [1, 2]. No single biochemical aberration unifies the diffuse nature of diabetic vascular disease. Hyperglycemia has been implicated, and yet glucose effects persist long after restoration of euglycemia. It is possible that acute fluctuations in glucose concentration have prolonged cell and tissue effects.

Copyright © 2007 by ASME

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