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Finite Element Modeling of the Lamina Cribrosa Microarchitecture in the Normal and Early Glaucoma Monkey Optic Nerve Head

[+] Author Affiliations
J. Crawford Downs, Michael D. Roberts, Claude F. Burgoyne

Devers Eye Institute, Portland, OR

Richard T. Hart

Ohio State University, Columbus, OH

Paper No. SBC2007-176193, pp. 357-358; 2 pages
doi:10.1115/SBC2007-176193
From:
  • ASME 2007 Summer Bioengineering Conference
  • ASME 2007 Summer Bioengineering Conference
  • Keystone, Colorado, USA, June 20–24, 2007
  • Conference Sponsors: Bioengineering Division
  • ISBN: 0-7918-4798-5
  • Copyright © 2007 by ASME

abstract

Glaucoma is the second leading cause of blindness in the US and is usually associated with elevated intraocular pressure (IOP). Glaucomatous damage is believed to occur at the optic nerve head (ONH) where the retinal ganglion cell axons pass through an opening in the back of the eye wall on their path to the brain. This opening is spanned by the lamina cribrosa, a fenestrated connective tissue structure that provides structural and nutritional support for the axons as they pass through the eye wall.

Copyright © 2007 by ASME

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