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CFD and VH-IVUS Biomechanical Analysis of Coronary Artery Disease With One Year Follow-Up

[+] Author Affiliations
David S. Molony, Don P. Giddens

Georgia Institute of Technology, Atlanta, GA

Lucas H. Timmins

Georgia Institute of Technology, Atlanta, GAEmory University School of Medicine, Atlanta, GA

Parham Eshtehardi

Albert Einstein College of Medicine, Bronx, NY

Habib Samady

Emory University School of Medicine, Atlanta, GA

Paper No. SBC2013-14313, pp. V01AT03A002; 2 pages
doi:10.1115/SBC2013-14313
From:
  • ASME 2013 Summer Bioengineering Conference
  • Volume 1A: Abdominal Aortic Aneurysms; Active and Reactive Soft Matter; Atherosclerosis; BioFluid Mechanics; Education; Biotransport Phenomena; Bone, Joint and Spine Mechanics; Brain Injury; Cardiac Mechanics; Cardiovascular Devices, Fluids and Imaging; Cartilage and Disc Mechanics; Cell and Tissue Engineering; Cerebral Aneurysms; Computational Biofluid Dynamics; Device Design, Human Dynamics, and Rehabilitation; Drug Delivery and Disease Treatment; Engineered Cellular Environments
  • Sunriver, Oregon, USA, June 26–29, 2013
  • Conference Sponsors: Bioengineering Division
  • ISBN: 978-0-7918-5560-7
  • Copyright © 2013 by ASME

abstract

Coronary atherosclerotic plaques are frequently focal lesions that have variable rates of progression. Wall shear stresses (WSS) create a number of responses in endothelial cells that can lead to the localization and progression of these lesions, and in vivo coronary segments with low WSS have been found to develop greater plaque progression than segments of higher WSS.

Copyright © 2013 by ASME

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