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Developing Brain and Heart Cells Respond Similarly to Altered Mechanical Loads

[+] Author Affiliations
Benjamen A. Filas, Philip V. Bayly, Larry A. Taber

Washington University, St. Louis, MO

Paper No. SBC2010-19029, pp. 773-774; 2 pages
doi:10.1115/SBC2010-19029
From:
  • ASME 2010 Summer Bioengineering Conference
  • ASME 2010 Summer Bioengineering Conference, Parts A and B
  • Naples, Florida, USA, June 16–19, 2010
  • Conference Sponsors: Bioengineering Division
  • ISBN: 978-0-7918-4403-8
  • Copyright © 2010 by ASME

abstract

Past studies have shown that the mechanical environment plays a critical role in regulating tissue development and function. For example, in the embryonic heart abnormal internal pressures cause morphological adaptation leading to aberrant morphogenesis [1]. Similarly, increasing luminal pressure in the early brain results in hyper-proliferation of the neuroepithelium [2]. Less is known, however, about how embryonic precursor cells quantitatively adapt to changes in loading, especially in vivo and across tissue types. These data would be valuable in determining the role of altered mechanical loads in congenital defects.

Copyright © 2010 by ASME
Topics: Stress , Brain

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